Biotech · Pharma · Darmstadt

Where Discovery
Becomes Therapy

Tavargenix translates the discovery of the TKTL1 gene into targeted therapies — for cancer, viral infections, and critical conditions like sepsis.

20+Years of Research
3Therapeutic Areas
2Breakthrough Genes
The Origin

A Discovery
That Changed
Everything

1995

The Gene is Found

Dr. Johannes Coy discovers TKTL1 — a gene that enables cancer cells to survive and proliferate without oxygen.

2003

The Mechanism Understood

Research reveals how TKTL1 drives the Warburg metabolism, giving tumors a unique survival advantage.

2006

Tavargenix Founded

Dr. Coy establishes Tavargenix GmbH in Darmstadt to translate fundamental science into therapeutic reality.

Today

Therapies in Development

Multiple programs advancing toward clinical application across oncology, virology, and global health.

In a quiet laboratory in the mid-1990s, a young biochemist named Johannes Coy made a finding that would define his life's work — and offer new hope to millions.

He identified a gene, TKTL1, that explained something scientists had long observed but never fully understood: why cancer cells thrive in low-oxygen environments where healthy cells cannot survive.

This wasn't just an academic discovery. TKTL1 is a key metabolic switch — one that cancer exploits to grow, spread, and resist treatment. Understanding it meant, for the first time, that we could target it.

"The question was never whether this discovery mattered. The question was: how quickly can we turn it into something that helps patients?"

That question became the founding mission of Tavargenix. Two decades later, it remains our North Star: to bridge the distance between fundamental discovery and life-changing therapy.

The TKTL1 Mechanism

TKTL1 is a transketolase-like enzyme that rewires cellular metabolism, enabling the Warburg effect — the hallmark of aggressive cancer growth.

Under normal conditions, cells rely on oxygen to produce energy. But TKTL1-overexpressing cells generate energy and growth building blocks even in hypoxic environments that should be inhospitable to life.

This metabolic flexibility makes TKTL1-positive cells extraordinarily resistant to conventional approaches. Targeting TKTL1 directly addresses this mechanism at its source.

01

Metabolic Reprogramming

TKTL1 overexpression shifts glucose metabolism from oxidative phosphorylation to a fermentative pathway, fueling rapid cell proliferation.

02

Hypoxia Survival

TKTL1-positive cells survive in low-oxygen tumor microenvironments — conditions that would normally trigger cell death.

03

Building Block Production

The enzyme produces ribose-5-phosphate essential for nucleotide and lipid synthesis, directly supporting uncontrolled tumor growth.

04

Therapeutic Target

By selectively inhibiting TKTL1 activity, Tavargenix's approach cuts the metabolic lifeline of cancer cells without harming healthy tissue.

Development Pipeline

From Molecule
to Medicine

Our lead program B-OT advances alongside a broader portfolio targeting three major disease areas where TKTL1 biology plays a critical role.

Discovery
Preclinical
Phase I
Approval

Pipeline status as of 2025 — subject to ongoing evaluation

B-OTLead Compound

B-OT: Targeting the Warburg Effect

B-OT is Tavargenix's lead therapeutic candidate — a small molecule designed to selectively inhibit the TKTL1-driven metabolic pathway. Preclinical studies demonstrate significant anti-tumor activity across multiple cancer models with a favorable safety profile. Phase II preparation is underway.

Program 01

Oncology

Targeting TKTL1-positive solid tumors across multiple indications. Initial focus on cancers with high TKTL1 expression and unmet therapeutic need.

Preclinical → Phase I
Program 02

Virology

Investigating the role of TKTL1 in viral replication mechanisms. Potential applications in chronic viral infections where metabolic intervention may be effective.

Discovery Stage
Program 03

Global Health

Developing accessible interventions for neglected diseases in low-resource settings, leveraging metabolic insights from the core TKTL1 platform.

Discovery Stage
Dr. Johannes Coy

Dr. Johannes Coy

Biochemist · Discoverer of TKTL1 · Founder & CEO

Dr. Johannes Coy is one of the rare scientists whose career-defining discovery has remained the foundation of a lifelong mission. In 1995, while conducting genetic research, he identified TKTL1 — a previously unknown gene that fundamentally alters how we understand cancer cell metabolism.

Rather than publish and move on, Dr. Coy spent the following decades investigating every dimension of TKTL1 biology: its role in tumor development, viral disease, and metabolic disorders. He holds numerous patents and has authored landmark publications in the field.

In 2006, he founded Tavargenix GmbH in Darmstadt to create the organizational vehicle his science needed — a focused, purpose-driven company capable of translating the TKTL1 discovery into therapies that reach patients.

Seine Geschichte
1995
Year of TKTL1 Discovery
30+
Patents Held
2006
Tavargenix Founded
Partnering

Build the Future
of TKTL1 Medicine

We are actively seeking partners who share our commitment to translating breakthrough science into accessible, effective therapies.

Pharmaceutical Partners

Co-development and licensing opportunities for our B-OT compound and pipeline assets across oncology indications.

Research Institutions

Academic and translational research collaborations to deepen understanding of TKTL1 biology and expand the therapeutic scope.

Investors & Funds

Strategic investment partnerships to accelerate clinical development and expand global health programs in underserved markets.

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